By Scott Buzby
Following SARS-CoV-2 infection, mitochondrial function remained impaired in the heart, liver and kidneys, despite observed recovery in the lungs, according to a human autopsy and animal tissue study.
“Upon SARS-CoV-2 infection of host cells, the viral copy number increases unchecked until the innate immune system is engaged, after which the viral copies progressively decline until the virus is eliminated,” Joseph Guarnieri, PhD, postdoctoral research fellow at the Center for Mitochondrial and Epigenomic Medicine at Children’s Hospital of Philadelphia, and colleagues wrote. Read the full article in Healio.